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  • Book
    edited by Stephen Gillam and A. Niroshan Siriwardena ; foreword by Iona Heath.
    Contents:
    Part 1. Background to the Quality and Outcomes Framework
    1. Introduction: development, impact and implications / Steve Gillam and A. Niroshan Siriwardena
    2. Where did the Quality and Outcomes Framework come from? / Martin Roland
    3. Developing indicators and the concept of QOF ability / Stephen Campbell and Helen Lester
    Part II. Impact of the Quality and Outcomes Framework
    4. Learning from the QOF: a review of existing research / Nicholas Steel and Sara Willems
    5. The public health impact / Anna Dixon, Artak Khachatryan and Tammy Boyce
    6. 'Smoke and mirrors'? Informatics opportunities and challenges / Maria Kordowicz and Mark Ashworth
    7. The impact of the QOF on practice organisation and service delivery / Kath Checkland and Stephen Harrison
    Part III. Practical aspects of the Quality and Outcomes Framework
    8. Getting the most out of the QOF / Chantal Simon and Anna Morton
    9. Does the patient always benefit? / Patricia Wilkie
    Part IV. Reflections on pay-for-performance and the Quality and Outcomes Framework
    10. Pay-for-performance schemes in primary care: what have we learnt? / Stephen Peckham and Andrew Wallace
    11. An international perspective on the basis of pay-for-performance / Barbara Starfield and Dee Mangin
    12. The Quality and Outcomes Framework: triumph of evidence or tragedy for personal care? / Steve Gillam and A. Niroshan Siriwardena.
    Print Access Request
    Location
    Version
    Call Number
    Items
    Books: General Collection (Downstairs)
    2011
    R728.5 .Q83 2011
    1
  • Article
    Zembala M, Mytar B, Popiela T, Asherson GL.
    Int J Cancer. 1977 May 15;19(5):605-13.
    The reactivity of peripheral blood lymphocytes from patients with advanced malignancy was assessed by mitogen-induced stimulation of protein synthesis as measured by 3H-leucine incorporation. It was confirmed that the lymphocyte response of patients was depressed. Furthermore, the lymphocytes of 15 out of 27 cancer patients, selected because of their low responses, inhibited the reactivity of normal lymphocytes in co-cultures. The lymphocytes from one patient with Hodgkin's disease were also inhibitory. In contrast, lymphocytes from healthy subjects, patients with chronic lymphocytic leukaemia, lymphosarcoma or multiple myeloma caused no suppression. Experiments with purified cell populations from patients with carcinoma indicated that purified T cells responded to mitogens while unseparated lymphocytes failed to respond and that the inhibitory activity was due to adherent cells, presumably monocytes. There was no evidence for B-cell-mediated suppression. However, in two cases inhibition was caused by isolated T cells of the patients and not by adherent cells. These experiments suggested that one mechanism for the depression of cell-mediated immunity seen in patients with advanced cancer may be the nonspecific suppresssion of certain T-cell functions by circulating monocytes.
    Digital Access Access Options