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  • Book
    edited by Alastair D. Burt, Bernard C. Portmann and Linda D. Ferrell.
    Contents:
    1. Anatomy, pathophysiology and basic mechanisms of disease / James M. Crawford and Alastair D. Burt
    2. Cellular and molecular techniques / Pierre Bedossa and Valerie Paradis
    3. Developmental abnormalities and liver disease in childhood / Bernard C. Portmann and Eve A. Roberts
    4. Genetic and metabolic liver disease / Richard J. Thompson, Bernard C. Portmann and Eve A. Roberts
    5. Disorders of iron overload / Alan C. Paterson and Antonello Pietrangelo
    6. Fatty liver disease : alcoholic and non-alcoholic / Elizabeth M. Brunt, Brent A. Neuschwander-Tetri and Alastair D. Burt
    7. Acute and chronic viral hepatitis / Neil D. Theise, Henry C. Bodenheimer Jr. and Linda D. Ferrell
    8. Other viral and infectious diseases and HIV-related liver disease / Sebastian B. Lucas, Sherif R. Zaki and Bernard C. Portmann
    9. Autoimmune hepatitis / M. Kay Washington and Michael P. Manns
    10. Diseases of the bile ducts / Yasuni Nakanuma, Yoh Zen and Bernard C. Portmann
    11. Diseases of the gallbladder / Jorge Albores-Saavedra and Arturo Angeles-Angeles --12. Vascular disorders / Ian R. Wanless and Weei-Yuarn Huang
    13. Hepatic injury due to drugs, herbal compounds, chemicals and toxins / James H. Lewis and David E. Kleiner
    14. Tumours and tumour-like lesions of the liver / Zachary D. Goodman, Luigi M. Terracciano and Aileen Wee
    15. Transplantation pathology / Stefan G. Hübscher and Andrew D. Clouston
    16. Systemic disease / Alberto Quaglia, Alastair D. Burt, Linda D. Ferrell, Bernard C. Portmann.
    Digital Access ScienceDirect 2011
  • Article
    Kahn MF, Peltier AP, Degraeve B, Mery JP, Morel-Maroger L, de Seze S.
    Ann Med Interne (Paris). 1977 Jan;128(1):1-8.
    The authors present 4 patients who had suffered for several years from isolated systemic scleroderma and in whom, after 3, 10, 19 and 22 years, clinical, biological and histological signs of DLE appeared. There was no current further progression of the scleroderma in two cases, at the time of onset of DLE. In the oter two cases, it had disappeared. All four patients had a serum cryoglobulin. In all cases the kidneys were involved histologically : extra membranous glomerulitis in one, segmental glomerulitis in a second, and diffuse progressive glomerulitis in the last two. The authors analyse the biological changes which accompanied the clinical transformation. One of the patients had anti-ribonucleoprotein antibodies. She died from severe renal involvement which would be against the good prognosis usually associated with the presence of this type of antibody.
    Digital Access Access Options