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  • Book
    Eran Zahavy ... [et al.], editors.
    Contents:
    Biomolecule/nanomaterial hybrid systems for nanobiotechnology
    Superresolution optical fluctuation imaging (SOFI)
    Application of nanoparticles for the detection and sorting of pathogenic bacteria by flow-cytometry
    Advancing nanostructured porous si-based optical transducers for label free bacteria detection
    Gold fibers as a platform for biosensing
    Surface-enhanced raman spectroscopy of organic molecules adsorbed on metallic nanoparticles
    Quantum dots and fluorescent protein FRET-based biosensors
    Semiconductor quantum dots as FRET acceptors for multiplexed diagnostics and molecular ruler application
    Assembly and microscopic characterization of DNA origami structures
    DNA nanotechnology
    Role of carbohydrate receptors in the macrophage uptake of dextran-coated iron oxide nanoparticles
    Toxicity of gold nanoparticles on somatic and reproductive cells
    Ultrasound activated nano-encapsulated targeted drug delivery and tumour cell poration
    Ultrasound mediated localized drug delivery
    Sonochemical proteinaceous microspheres for wound healing
    Alendronate liposomes for antitumor therapy: activation of [gamma] [delta] T cells and inhibition of tumor growth.
    Digital Access Springer 2012
    Access via Advances in experimental medicine and biology ; 2012; 733
    Location
    Version
    Call Number
    Items
    Advances in experimental medicine and biology ; 2012; 733
    c2012
  • Article
    Fisher MS, Kripke ML.
    Proc Natl Acad Sci U S A. 1977 Apr;74(4):1688-92.
    Chronic irradiation of mice with ultraviolet (UV) light produces a systemic alteration of an immunologic nature. This alteration is detectable in mice long before primary skin cancers induced by UV light begin to appear. The alteration results in the failure of UV-irradiated mice to reject highly antigenic, transplanted UV-induced tumors that are rejected by unirradiated syngeneic recipients. The immunologic aspect of this systemic alteration was demonstrated by transferring lymphoid cells from UV-irradiated mice to lethally x-irradiated recipients. These recipeints were unable to resist a later challenge with a syngeneic UV-induced tumor, whereas those given lymphoid cells from normal donors were resistant to tumor growth. Parabiosis of normal mice with UV-irradiated mice, followed by tumor challenge of both parabionts with a UV-induced tumor, resulted in the growth of the challenge tumors in both UV-irradiated and unirradiated mice. Splenic lymphocytes from tumor-implanted UV-treated mice were not cytotoxic in vitro against UV-induced tumors, whereas under identical conditions cells from tumor-implanted, unirradiated mice were highly cytotoxic. Our findings suggest that repeated UV irradiation can circumvent an immunologic mechanism that might otherwise destroy nascent UV-induced primary tumors that are strongly antigenic.
    Digital Access Access Options