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  • Book
    Óliver Marín-Peña, editor.
    Contents:
    pt. 1. Concept and physical exam in FAI
    1. Historical Evolution of the Concept of Femoroacetabular Impingement as a Cause of Hip Osteoarthritis / Martin Lavigne, Laffosse Jean-Michel and Vendittoli Pascal-André
    2. Mechanism of Femoroacetabular Impingement / Martin Beck, Slaman Chegini, Stephen Ferguson and Harish S. Hosalkar
    3. Physical Exam in FAI / Óliver Marín-Peña
    pt. 2. Imaging in FAI
    4. X-Ray Examination in FAI / Klaus A. Siebenrock and Philipp Henle
    5. MRI/CT in FAI / Kawan S. Rakhra
    6. Future Strategies for the Assessment of Cartilage and Labral Lesions in Femoroacetabular Impingement / Ara Kassarjian, Luis Cerezal and Eva Llopis
    pt. 3. FAI Surgical Treatment
    7. Evidence-Based Medicine in the Treatment of Femoroacetabular Impingement / Ricardo Larraínzar Garijo, R. García-Bógalo and E. Díez-Nicolás
    8. Bone Resection: First Step for Treatment, How Much Is Too Much? / Rodrigo Mardones and Fernando Nemtala
    9. Open Surgical Treatment of FAI: Safe Surgical Dislocation of the Femoral Head / Michael Leunig, Anil Ranawat, Martin Beck and Reinhold Ganz
    10. Mini-Anterior Approach / Manuel Ribas and Óliver Marín-Peña
    11. Arthroscopic Treatment of FAI: Position, Portals, and Instrumentation / Victor M. Ilizaliturri
    12. Normal and Pathological Arthroscopic View in Hip Arthroscopy / Damian Griffin and Shanmugam Karthikeyan
    13. What Goes on During the Learning Curve? / Luís Perez-Carro and Marc Tey
    14. My Experience of Hip Arthroscopy in the Lateral Position / Alexandros P. Tzaveas and Richard N. Villar
    15. Arthroscopic Treatment of FAI: Supine Position, My First Option / Marcelo Quieroz, Katrina Dela Torre and Bryan T. Kelly
    16. Complications and Revision Surgery in Hip Arthroscopy / Bruno G. S. e Souza and Marc J. Philippon
    17. Combined Techniques in FAI: Hip Arthroscopy Followed / Mini-Anterior Approach / Nader A. Nassif and John C. Clohisy
    18. Femoroacetabular Impingement Management Through a Mini-Open Anterior Approach and Arthroscopic Assistance: Technics and Mid-Term Results / Frédéric Laude and Elhadi Sariali
    pt. 4. F.A.I. and Hip Dysplasia
    19. Differentiating FAI from Dysplasia / Wadih Y. Matar and Javad Parvizi
    20. Advances in PAO Surgery: The Minimally Invasive Approach / Anders Troelsen and Kjeld Søballe
    21. Retroverted PAO or Rim Trimming in the Dysplastic Hip with FAI / Philipp Henle and Klaus A. Siebenrock
    pt. 5. Hip arthroplasty in young adult
    22. Current Arthroplasty Options in the Young Adult Hip / Eduardo García Cimbrelo, Eduardo Garcia-Rey and Ana Cruz-Pardos
    23. Resurfacing Arthroplasty for Femoroacetabular Impingement / Matías J. Salineros and Paul E. Beaulé
    24. How to Do Resurfacing in Hip Dysplasia / Koen De Smet
    pt. 6. Postoperative management (POM)
    25. Rehabilitation Following Femoroacetabular Impingement Surgery / Lafayette de Azevedo Lage
    26. Postoperative Management of Hip Resurfacing / Alfonso Valles and Carlos Gebhard
    pt. 7. Outcome in FAI Treatment
    27. Clinical Scores in Femoroacetabular Impingement / Néstor Moreno and Óliver Marín-Peña.
    Digital Access Springer 2012
  • Article
    Scubon-Mulieri B, Parsons RL.
    J Gen Physiol. 1977 Apr;69(4):431-47.
    The time course of carbachol-induced desensitization onset and recovery of sensitivity after desenitization have been compared at the frog neuromuscular junction. The activation-desensitization sequence was determined from input conductance measurements using potassium-depolarized muscle preparations. Both desensitization onset and recovery from desensitization could be adequately described by single time constant expressions, with tauonset being considerably shorter than taurecovery. In nine experiments, tauonset was 13+/-1.3 s and taurecovery was 424+/-51 s with 1 mM carbachol. Elevating the external calcium or carbachol concentration accelerated desensitization onset without changing the recovery of sensitivity after equilibrium desensitization. Desensitization onset was accelerated by a prior activation-desensitization sequence to an extent determined by the recovery interval that followed the initial carbachol application. The time course of return of tauonset was closely parallel to, but slower than the time course of recovery of sensitivity. These results are consistent with a cyclic model in which intracellular calcium is a factor controlling the rate of development of desensitization.
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