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  • Book
    Johng S. Rhim, Anatoly Dritschilo, Richard Kremer, editors.
    Contents:
    Intro; Preface; Acknowledgments; Contents; Part I: Mechanisms of Tumor Progression; Mechanisms Underlying Metastatic Pancreatic Cancer; PDAC Metastasis and Outcomes; Models of Metastasis; Genetically Engineered Mouse Models of Metastatic PDAC; Orthotopic Models of Pancreatic Cancer Metastasis; Mechanisms of Metastasis; EMT-MET Axis and Cellular Plasticity; Metastatic Organotropism: The Role of Exosomes and EMT Modulators; Epigenetic and Post-Transcriptional Regulators of EMT and Metastasis; Concluding Remarks; References; Current Perspectives on Nasopharyngeal Carcinoma; Introduction Historic PerspectiveCurrent Perspectives; Epidemiology; Incidence Rate; Impact of Human Migration on NPC Incidents; Risk Factors; Epstein Barr Virus (EBV); Single Nucleotide Polymorphisms (SNPs); Intake of Certain Foods; Model System for Investigating Nasopharyngeal Carcinoma; Potential Use of Zebrafish for Modeling Human NPC; Immunotherapy for Nasopharyngeal Carcinoma; Immunotherapy: General Concepts; Relevant Cancer Mouse Models for Preclinical Investigation of Immunotherapy; Immunotherapeutic Approaches for Cancer Treatment; Immunotherapy in NPC; Concluding Remarks; References An In Vitro Model of Triple-Negative Breast Cancer17€-Estradiol Induces Transformation and Tumorigenesis in Human Breast Epithelial Cells; Epithelial to Mesenchymal Transition in Human Breast Epithelial Cells Transformed by 17-Beta-Estradiol; Developing a Unique Model of Triple Negative Breast Cancer; Chromatin Remodeling During Human Breast Epithelial Cell Transformation; Concluding Remarks; References; Emerging Role of Novel Biomarkers of Ly6 Gene Family in Pan Cancer; Introduction; Expression of Ly6D, E, H, and K Genes in Normal Tissues; Ly6D; Ly6E; Ly6H; Ly6K Expression of Ly6D, E, H, and K Genes in Tumor TissuesLy6D; Ly6E; Ly6H; Ly6K; Mechanisms Associated Ly6D, E, H, and K Gene Family; Ly6D; Ly6E; Ly6H; Ly6K; Summary; References; The Oncoprotein Gankyrin/PSMD10 as a Target of Cancer Therapy; Isolation of Gankyrin from Hepatocellular Carcinoma; Enhanced Degradation of RB (Retinoblastoma-Associated Protein) and p53 (Cellular Tumor Antigen p53) by Gankyrin; Gankyrin as a Killer of Multiple Tumor Suppressor Proteins; Animal Models Overexpressing Gankyrin in the Liver Importance of Non-Parenchymal Cells in Carcinogenesis as Demonstrated by Gankyrin-Knockout MiceGankyrin as a Promising Therapeutic Target; Experimental Anti-Gankyrin Agents; Conclusion; References; Contributing Roles of CYP2E1 and Other Cytochrome P450 Isoforms in Alcohol-Related Tissue Injury and Carcinogenesis; Introduction; Updated Mechanisms of Ethanol-Mediated Carcinogenesis; Contributing Roles of CYP2E1 and Other P450 Isoforms in Tissue Injury and Cancer Development; Multiple Regulations of CYP2E1 and Alcohol-Related Tissue Injury and Carcinogenesis Distribution of CYP2E1 and Carcinogenesis in Extra-Hepatic Tissues
    Digital Access Springer 2019
  • Book
    George Wolberg.
    Summary: This best-selling, original text focuses on image reconstruction, real-time texture mapping, separable algorithms, two-pass transforms, mesh warping, and special effects. The text, containing all original material, begins with the history of the field and continues with a review of common terminology, mathematical preliminaries, and digital image acquisition. Later chapters discuss equations for spatial information, interpolation kernels, filtering problems, and fast-warping techniques based on scanline algorithms. Nielsen 9780818689444 20160528
    Print c1990
  • Article
    Epstein CJ, Golbus MS.
    Annu Rev Med. 1978;29:117-28.
    Prenatal diagnosis has proven highly effective in assessing the status of fetuses at risk of all cytogenetic and of several biochemical and structural disorders with genetic etiologies. The original methodology, based on the cytogenetic and biochemical analysis of cultured amniotic fluid cells, has now been complemented by a wide variety of new techniques. These include several methods for fetal visualization (sonography, fetoscopy, x ray), sampling of fetal blood, and analysis of very small quantities of material. Additional approaches, based on these and other technologies, are likely to permit the prenatal diagnosis of an ever increasing number of genetic disorders. At the same time, the number of pregnancies monitored will increase as greater resources become available and as screening programs identify couples at risk of having genetically abnormal children prior to the birth of an affected child. Prenatal diagnosis is already a powerful means of preventing the birth of individuals with significant genetic defects, thereby sparing both individuals and society from the burdens that such disorders produce. In the future, it is likely to be even more effective.
    Digital Access Access Options