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- Book[edited by] Hanno W. Kirk.Summary: "This thoroughly updated second edition of Restoring the Brain is the definitive book on the theory and the practice of Infra-Low Frequency brain training. It provides a comprehensive look at the process of neurofeedback within the emerging field of neuromodulation and essential knowledge of functional neuroanatomy and neural dynamics to successfully restore brain function. Integrating the latest research, this thoroughly revised edition focuses on current innovations in mechanisms-based training that are scalable and can be deployed at any stage of human development. Included in this edition are new chapters on clinical data and case studies for new applications; using neurofeedback for early childhood developmental disorders; integrating neurofeedback with psychotherapy; the impact of low-frequency neurofeedback on depression; the issue of trauma from war or abuse; and physical damage to the brain. Practitioners and researchers in psychiatry, medicine, and behavioral health will gain a wealth of knowledge and tools for effectively using neurofeedback to recover and enhance the functional competence of the brain"-- Provided by publisher.
Contents:
Cover
Half Title
Title Page
Copyright Page
Dedication
Table of Contents
Foreword
Preface
Notes on Editor and Contributors
Acknowledgments
Introduction
Part I: Development, History, and Theory of Neurofeedback
1. Changing the Paradigm from Neurochemical to Neuroelectrical models
1.1 The Early Days of Speculation
1.2 The Soups and the Sparks
1.3 The Neurochemical Paradigm
1.4 Moving to a New Paradigm
1.5 Traditional Wave Bands
1.6 Conclusion
Notes
2. History of Neurofeedback
2.1 The Scientific Antecedents of Neurofeedback 2.2 The Foundations of Neurofeedback in Animal Studies
2.3 Evaluation of SMR Reinforcement with Human Subjects
2.4 Neurofeedback in Application to Addictions
2.5 Slow Cortical Potential Training
2.6 New Departures in Neurofeedback
2.7 Status of the Principal Approaches to Neurofeedback
2.8 Toward a New Departure in Feedback
2.9 The Development of Infra-Low Frequency Training
2.10 Toward an Understanding of Covert, Continuous ILF Neurofeedback
2.11 A Summary Perspective
Notes
3. Toward a Theory of Infra-Low Frequency Neurofeedback
3.1 Introduction 3.2 Categories of Brain Dysfunction
3.3 The Trauma Model
3.4 Mechanisms of Regulation: Historical Roots of the Slow Cortical Potential
3.5 ILF Neurofeedback in the Frequency Domain: The Frequency Rules
3.6 A Resonance Phenomenon
3.7 Implications for Inter-hemispheric Coordination
3.8 Foundational Research in Characterization of ILF Neurofeedback
3.9 Summary and Conclusion
Notes
4. Astrocytes and Infra-Low Frequencies
4.1 Introduction
4.2 Rhythmicity of the Brain and Energy Cycles
4.3 Implications for Neurofeedback
Notes Part II: Neurofeedback in Clinical Practice Settings
5. Neurofeedback in Clinical Practice
5.1 O verview of the Clinical Process and Development of Individual Treatment Plans
5.2 Combining the ILF Approach with Proven Older Neurofeedback Methods
5.3 Closing Remarks for Neurofeedback Clinicians
Notes
6. Neurofeedback in an Integrative Medical Practice
6.1 Introduction
6.2 Assessment and Screening
6.3 Case Presentations
6.4 Conclusion
Notes
7. Neurofeedback in Combination with Psychotherapy
7.1 Overview 7.2 Case Study #1: Bipolar I Disorder, Postpartum Mixed Episode
7.3 Case Study #2: Severe Generalized Anxiety Disorder with Severe Alcohol Use Disorder
7.4 Conclusion
Notes
8. Remediating Brain Instabilities in a Neurology Practice
8.1 Introduction
8.2 Case Vignettes
8.3 Exploiting Neuroplasticity
8.4 Patient Selection
8.5 Prescribing Brain Training Protocols
8.6 Viewpoints: Neurofeedback Practice in a Neurology Office
8.7 Summary Statement
Notes
Part III: Specific Applications Areas of Neurofeedback - ArticleJörstad S, Kvernes S.Acta Pathol Microbiol Scand C. 1978 Oct;86C(5):221-6.Uraemic plasma and normal serum were separated into fractions of different molecular sizes by means of molecular filtration through Pellicon filters and liquid chromatography on Sephadex G-15. The amount of substance was considerably higher in uraemic fractions than in comparable normal fractions. When normal human mononuclear phagocytes were cultured in the presence of the uraemic fraction containing the largest molecules, detachment of phagocytes from the glass coverslips was significantly increased. The digestion capacity of the remaining cells was decreased. The results indicate that substances in uraemic plasma responsible for the impaired function of human mononuclear phagocytes cultured in vitro consist of molecules of a molecular weight higher than 10 000.