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- BookVladimír Beneš, Ondřej Bradáč, editors.Contents:
Introduction
Motivation to this study
AVM definition and angioarchitecture
Feeding arteries
Associated aneurysms
Nidus component
Draining veins
Genetics of arteriovenous malformations
Epidemiology of AVM
Physiology of arteriovenous malformations
Arterial side
Steal phenomenon
Normal perfusion pressure breakthrough
Venous side
Occlusive hyperemia
Compartmental model of AVM nidus
AVM presentation
Hemorrhagic presentation
Morbidity and mortality of AVM related intracranial haemorrhage
Epilepsy presentation
Focal neurologic deficit (FND)
Natural course
AVM grading
Surgical AVM grading
Radiosurgical AVM grading
Imaging
Catheter angiography
Magnetic resonance imaging
Computed tomography
Treatment for brain AVMs
Literature series
Surgery
Endovascular embolization
Stereotactic radiosurgery
Combined treatment
ARUBA study comments.Digital Access Springer 2017 - ArticleMilch H, Czirók E, Madár J, Semjén G.Acta Microbiol Acad Sci Hung. 1977;24(2):127-37.Escherichia coli O78: K80 strains isolated from an outbreak of meningitis, sepsis and enteritis in infants, were compared with O78: K80 strains from sporadic cases of enteritis, healthy carriers and animals. The strains were uniform in antigenic structure and phage pattern but differed in colicinogenicity. The epidemic strains and calf-pathogenic cultures produced colicin V, the remaining isolates were characterized by other types of colicin or were not colicinogenic. Col V+ strains multiplied in the mouse peritoneal cavity more readily and killed the animals at significantly lower doses than did col V- strains. One half of antibiotic resistant O78: K80 strains carried R factor. The spread of R factor could be followed by phage restriction experiments.