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  • Book
    Ann E. Kearns, Robert A. Wermers, editors.
    Contents:
    Asymptomatic Primary Hyperparathyroidism
    Severe Primary Hyperparathyroidism
    Nephrolithiasis in Primary Hyperparathyroidism
    Primary Hyperparathyroidism and Osteoporosis
    Parathyroid Hormone Measurement Considerations in Primary Hyperparathyroidism
    Parathyroidectomy Outcomes and Pathology in Primary Hyperparathyroidism
    Localization Considerations in Persistent Primary Hyperparathyroidism
    Ectopic Parathyroid Adenoma
    Parathyroid Surgery in Multiple Endocrine Neoplasia 1
    Parathyroid Carcinoma
    Primary Hyperparathyroidism Due to Parathyromatosis
    Familial Hypocalciuric Hypercalcemia
    Medical Management of Primary Hyperparathyroidism
    Primary Hyperparathyroidism in Children and Adolescents
    Primary Hyperparathyroidism in Pregnancy
    Primary Hyperparathyroidism: Association with Co-Existent Secondary Causes of Hypercalcemia
    Medication Considerations in Hypercalcemia and Hyperparathyroidism
    Normocalcemic Primary Hyperparathyroidism
    Secondary Hyperparathyroidism
    Tertiary Hyperparathyroidism.
    Digital Access Springer 2016
  • Article
    Briquet M.
    Biochim Biophys Acta. 1977 Feb 07;459(2):290-9.
    Evidence for the existence of mediated transport of pyruvate and lactate in isolated mitochondria of Saccharomyces cerevisiae is presented. 1. The mitochondrial oxidation of pyruvate is specifically inhibited by the monocarboxylic oxoacids alpha-ketoisocaproate and by alpha-cyano-3-hydroxycinnamate, while pyruvate and malate dehydrogenases activities are not inhibited. 2. The stimulation of the mitochondrial oxidations of succinate, alpha-ketoglutarate and citrate by pyruvate are also inhibited by alpha-cyano-3-hydroxycinnamate. 3. The [14C]pyruvate uptake by yeast mitochondria follows saturation kinetics and is completely inhibited by alpha-cyano-3-hydroxycinnamate. 4. Large amplitude passive swellings of mitochondria of the wild type and of cytoplasmic rho- and rho-n mutants are induced by isoosmotic ammonium pyruvate and lactate. These pH-dependent swellings are inhibited by alpha-cyano-3-hydroxycinnamate suggesting that the carrier system is not coded by mitochondrial DNA.
    Digital Access Access Options