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  • Book
    edited by Connie Watkins Bales, Julie L. Locher, Edward Saltzman.
    Summary: This is the new and fully revised third edition of the well-received text that is the benchmark book in the field of nutrition and aging. The editors (specialists in geriatric nutrition, medical sociology, and clinical nutrition, respectively) and contributors (a panel of recognized academic nutritionists, geriatricians, clinicians, and other scientists) have added a number of new chapters and have thoroughly updated the widely acclaimed second edition. This third edition provides fresh perspectives and the latest scientific and clinical developments on the interaction of nutrition with age-associated disease and provides practical, evidence-based options to enhance this at-risk population?s potential for optimal health and disease prevention. Chapters on a wide range of topics, such as the role of nutrition in physical and cognitive function, and coverage of an array of clinical conditions (obesity, diabetes, heart failure, cancer, kidney disease, osteoporosis), compliment chapters on food insecurity, anti-aging and nutritional supplements, making this third edition uniquely different from previous editions. Handbook of Clinical Nutrition and Aging, Third Edition, is a practical and comprehensive resource and an invaluable guide to nutritionists, physicians, nurses, social workers and others who provide health care for the ever-increasing aging population.

    Contents:
    Part I. Overarching Issues for Nutritional Well-Being in Later Life
    1. Nutrition Interventions for Aging Populations
    2. Systematic Reviews in the Field of Nutrition
    3. Nutrition Assessment
    Part II. Fundamentals of Nutrition and Geriatric Syndromes
    4. Nutrition and the Aging Eye
    5. Nutrition and Oral Health: A Two-Way Relationship
    6. Loss of Muscle Mass and Muscle Strength in Obese and Non-Obese Older Adults
    7. Muscle Metabolism, Nutrition, and Functional Status in Older Adults
    8. Nutrition in the Prevention and Treatment of Cognitive Decline
    9. Food Insecurity and Hunger Among Older Adults
    Part III. Common Clinical Conditions
    10. Obesity in Older Adults and Strategies for Weight Management
    11. Nutrition and Lifestyle Change in Older Adults with Diabetes Mellitus and Metabolic Syndrome
    12. Preventive Cardiology: Counseling Older At-Risk Adults on Nutrition
    13. Chronic Heart Failure
    14. The Relationship of Nutrition and Pressure Ulcers
    15. Nutrition Support in Solid Tumor Cancer Patients
    16. Nutrition and Chronic Kidney Disease
    17. Nutritional Concerns in Osteoporosis
    18. Dementia-related Mealtime Difficulties: Assessment and Management in the Long-Term Care Setting
    19. Nutrition at the End of Life
    Part IV. Contemporary Diet-Focused Concerns
    20. Anti-Aging Effects of Nutritional Modification: The State of the Science on Calorie Restriction
    21. High-Risk Nutrients in the Aging Population
    22. Physical Activity and Exercise: Important Complements to Nutrition in Older Adults
    23. Dietary Supplements in Older Adults.
    Digital Access Springer 2015
  • Article
    Kurrle R, Wagner H, Röllinghoff M, Rott R.
    Eur J Immunol. 1979 Feb;9(2):107-11.
    This study deals with the requirements for target cell recognition by influenza A virus-specific cytotoxic T lymphocytes (CTL). H-2-identical cells were incubated with infectious or UV light-inactivated influenza A virus expressing either cleaved or uncleaved hemagglutinin (HA). Thereafter, the treated cells were tested in a 4-h 51Cr assay for susceptibility to CTL-mediated cytolysis. Regardless whether the influenza virus was infectious, virions expressing cleaved HA were efficient in target cell formation. In contrast, cells incubated with either active or UV-inactivated virions expressing uncleaved HA were not lysed by virus-specific CTL. Yet, after mere trypsin-mediated cleavage of the HA of cell-absorbed viroins, strong cytolysis could be observed. On the other hand, solubilization of the envelope lipid bilayer by ethylether abolished the capacity of the remaining HA to induce target cell formation. The results clearly suggest that mere absorption of virions to the membrane of cells, which is performed by virus with uncleaved HA, is insufficient for target cell formation. For this, both cleaved HA and an intact envelope appear to be crucial. We conclude that fusion of the virion into the cell membrane is essential for target cell formation.
    Digital Access Access Options