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  • Book
    Eleanor H. Simpson, Peter D. Balsam, editors.
    Contents:
    Part 1
    THE NEUROBIOLOGY OF COMPONENTS OF MOTIVATIONAL DRIVE
    Regulation of the Motivation to Eat
    Sexual Motivation Opposed by the Influences of Stress
    Motivational Forces Driving Social Behaviours
    The Roles of Wanting and Liking in Motivating Behaviour, Including Gambling, Food and Drug Addiction. Circadian Regulation of Motivated Behaviour
    How Negative Forces Influence Motivations Part 2
    NEURAL MEASURES AND CORRELATES OF MOTIVATION SIGNALS AND COMPUTATIONS
    Neural Computations of Postive and Negative Reward Value
    The Dopaminergic Regulation of Motivated Behaviour
    Neural Modulators other than Dopamine that Regulate Motivated Behaviours
    Neural Correlates of Motivation Signals in Humans
    Motivational Forces in Decision Making Processes
    Part 3
    APATHY AND PATHOLOGICAL DEFICITS IN MOTIVATION
    The Identification of Phenotypes and Biomarkers in Depression
    Defining Motivation Deficits in Schizophrenia and Relating them to Clinical Symptoms
    Dissecting the Behavioural components of deficits in motivation in Patients with Schizophrenia
    How Deficits in Motivation are Similar and Different in Depression and Schizophrenia
    Modeling Motivation Deficits in Rodents
    Part 4
    ADDICTION AND THE APTHOLOGICAL MISDIRECTION OF MOTIVATED BEHAVIOUR
    Motivation in Overdrive: Substance Abuse Disorder
    Insight into Gambling motivation from Animal Models
    Part 5
    DEVELOPMENTS IN TREATMENTS FOR MOTIVATION PATHOLOGIES
    The Role of Motivation in Schizophrenia Cognitive Remediation
    The Impact of Regulating Focus on Motivated Behaviour
    Insights into Gambling Motivation from Animal Models
    Exploring Pharmacological Agents in Animal Models.
    Digital Access Springer 2016
  • Book
    Contents:

    v. 1. Barnacles.--
    v. 2. Polyzoa.--
    v. 3. Serpulids.--
    v. 4. Ascidians.--
    v. 5. Marine sponges.--
    v. 6. Algae.--
    v. 7. Hydroids.
    Print 1963]-
  • Article
    Gelfand DH, Rudo N.
    J Bacteriol. 1977 Apr;130(1):441-4.
    Co-transduction experiments using P1-mediated reciprocal and three-factor crosses have been used to map two mutations affecting the aspartate and aromatic amino acid aminotransferases of Escherichia coli. tyrB-, which inactivates the tyrosine-repressible component of these activities is co-transducible with metA and malB; the gene order is metA-malB-tyrB. aspC-, which inactivates the nonrepressible aminotransferase with high activity for aspartate, maps between and is co-transducible with serC and pyrD.
    Digital Access Access Options