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  • Book
    Harold G. Koenig, Dana E. King, Verna Benner Carson.
    Summary: "Completely revises and updates the first edition, ... surveys the historical connections between religion and health and grapples with the distinction between the terms ''religion'' and ''spirituality'' in research and clinical practice. It reviews research on religion and mental health, as well as extensive research literature on the mind-body relationship, and develops a model to explain how religious involvement may impact physical health through the mind-body mechanisms. It also explores the direct relationships between religion and physical health, covering such topics as immune and endocrine function, heart disease, hypertension and stroke, neurological disorders, cancer, and infectious diseases; and examines the consequences of illness including chronic pain, disability, and quality of life ...[The] authors are physicians: a psychiatrist and geriatrician, a primary care physician, and a professor of nursing and specialist in mental health nursing"--Provided by publisher.

    Contents:
    I. Background. A history of religion, medicine, and health care
    Definitions
    II. Debating religion's effects on health. Religion : good or bad?
    Coping with stress
    Religion and coping
    III. Research on religion and mental health. Well-being and positive emotions
    Depression
    Suicide
    Anxiety disorders
    Psychotic disorders
    Alcohol and drug use
    Delinquency and crime
    Marital instability
    Personality and personality disorder
    Understanding religion's effects on mental health
    IV. Research on religion and physical health. Heart disease
    Hyptertension
    Cerebrovascular disease
    Alzheimer's Disease and dementia
    Immune functions
    Endocrine functions
    Cancer
    Mortality
    Physical disability
    Pain and somatic symptoms
    Health behaviors
    Disease prevention
    V. Understanding the religion-physical health relationship. Psychological, social, and behavioral pathways
    Conclusions
    Appendix. Studies on religion and health (by health outcome).
    Print Access Request
    Location
    Version
    Call Number
    Items
    Books: General Collection (Downstairs)
    2012
    BL65.M4 K646 2012
    1
  • Article
    Schuetz AW, Samson D.
    J Exp Zool. 1979 Nov;210(2):307-19.
    Cycloheximide induced a complex series of alterations in the cortical cytoplasm of amphibian (Rana pipiens) oocytes undergoing steroid induced nuclear and cytoplasmic maturation in vitro. The morphological changes were described and the role of nuclear-cytoplasmic interactions in the induction of these changes was investigated in intact, enucleated and enucleated-reinjected oocytes. Three stages of cortical changes were ascertained on the basis of: localized alterations at the animal pole, redistribution of pigment and localized contractility (furrow formation) primarily along the animal:vegetal pole axis. The extent and type of cortical alterations varied depending upon the time at which oocytes were examined following hormonal stimulation and cycloheximide treatment. Cycloheximide did not produce cortical alterations in non-hormone treated oocytes nor in steroid treated oocytes until after germinal vesicle breakdown. Nuclear and cytoplasmic maturation and the appearance of cortical alterations were all inhibited when cycloheximide was added to oocytes at the time of steroid treatment. Cycloheximide induction of cortical alterations occurred only after the inhibitor was no longer effective in preventing germinal vesicle breakdown. Enucleated oocytes underwent cytoplasmic maturation in response to the steroid but exhibited no cortical alterations following the delayed addition of cycloheximide. Simultaneous administration of cycloheximide and steroid to enucleated oocytes inhibited cytoplasmic maturation and all observable cortical alterations. Reinjection of nuclear material into enucleated oocytes restored the ability of cycloheximide to induce cortical alterations following steroid induction of cytoplasmic maturation. Without steroid treatment, such reinjected oocytes did not exhibit cortical changes in response to cycloheximide. The data demonstrate that the nucleus is required for and contains a factor(s) which controls the cycloheximide response and post-maturation differentiation of the oocyte. The maturational changes in the cortical cytoplasm appear to be dependent on the intermixing of the germinal vesicle nucleoplasm materials with mature cytoplasm following germinal vesicle breakdown. The results further suggest that the cortical effects of cycloheximide are dependent upon the initiation of protein synthesis during this period of oocyte development. The significance of these observations and experimental studies are discussed in relation to current understanding of the molecular mechanisms controlling meiosis induction and the composition of the germinal vesicle.
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