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  • Book
    editors: Jeanne Leffers, Claudia M. Smith, Katie Huffling, Ruth McDermott-Levy, Barbara Sattler.
    Contents:
    Introduction
    Unit I: Why nursing. Why nurses are involved with environmental health
    Environmental health and families/homes
    Environmental health in the school setting
    Environmental health nursing at the community level
    Faith community: an environmental partner
    Spotlight on nurses
    Desired environmental health competencies for registered nurses
    Unit II: Harmful environmental exposures and vulnerable populations. Harmful environmental exposures and vulnerable populations
    Vulnerable populations - anticipatory guidance
    Immigrants and refugees as a vulnerable population
    Unit III: Environmental health sciences. Ecology, ecosystems, and watersheds
    Introduction to toxicology
    Carcinogens
    Criteria air pollutants
    Chemicals of concern: flame retardants
    Health effects of heavy metals
    Environmental public health tracking
    Introduction to risk assessment in environmental health
    Unit IV: Practice settings. Nurse exposure in work
    Hazardous exposures in healthcare
    Pharmaceutical waste
    Green team
    Unit V: Sustainable communities. Introduction to Tox Town
    Green building
    Green cleaning in homes
    Transportation and health implications
    Brownfields and nursing implications
    Antibiotic use in agriculture: public and environmental health impact
    Environmental justice
    Unit VI: Climate change. Climate and health
    Unit vii: energy
    Introduction to the ANA energy resolution
    Unconventional natural gas development and nursing
    Unit VIII: Advocacy. Using nursing process to guide advocacy for environmental health
    Coalition building: a powerful political strategy
    Case study in environmental health advocacy
    Chemical policy reform - toxic chemicals in the environment: efforts to control and regulate
    Anatomy of a legislative meeting
    Unit IX: Research. Maria Amaya, PhD, RNC, WHNP-BC
    Adelita Cantu, PhD, RN
    Viki Chaudrue, EdD, MSN, RN
    Rosemary Chaudry, PhD, RN, MHA, MPH, PHCNS-BC
    Linda McCauley, RN, PhD, FAAN, FAAOHN
    Ruth McDermott-Levy, PhD, MPH, RN
    Barbara Polivka, PhD, RN
    Elizabeth C Schenk, PhD, MHI, RN
    Unit X: Conclusion.
    Digital Access R2Library 2015
  • Article
    Ahlstedt S, Carlsson B, Fällström SP, Hanson LA, Holmgren J, Lidin-Janson G, Lindblad BS, Jodal U, Kaijser B, Solh-Akerlund A, Wadsworth C.
    Ciba Found Symp. 1977 Apr 26-28(46):115-34.
    Ingestion of Escherichia coli O83 bacteria by adults resulted in a transient irregular colonization leading to a serum antibody response in only four out of 14 cases examined. In all of three pregnant women, however, IgA antibodies against E. coli O83 antigen were released from colostral cells after similar bacterial ingestion although no serum antibody response was noted. The findings indicate a link between the antigenic exposure to the gut and secretory antibodies of the IgA class, presumably locally formed in the mammary gland. Antibodies of the secretory IgA class registered in colostrum may, at least partly, reflect the antigenic exposure of the gut. These antibodies are probably important in protecting against E. coli infections in the neonate, as suggested by the findings of antibodies in human milk against O and K antigens of non-enteropathogenic as well as enteropathogenic serotypes of E. coli. Furthermore, in milk of women from low socio-economic groups in Pakistan, neutralizing antibodies were present against enterotoxins of E. coli bacteria and occasionally against Vibrio cholerae enterotoxins. In addition, secretory IgA antibodies against food proteins were detected in human milk. This suggests that intestinal exposure to such antigens could stimulate a local immune response in the gut resulting in triggered lymphoid cells homing to the mammary gland. These human milk secretory IgA antibodies against bovine milk proteins may help to prevent cow's milk allergy in infants on mixed feeding, since these infants tend to have a lower serum antibody response to cow's milk proteins than infants fed mostly artificially. Furthermore, children suffering from cow's milk protein intolerance and gluten enteropathy may have higher serum levels of antibody to cow's milk protein antigens than normal children, possibly reflecting increased permeability of the intestinal mucosa for various antigens.
    Digital Access Access Options