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- BookSanjeev Sabharwal, editor.Contents:
Part I: General Principles and Techniques
Etiology of Lower Limb Deformity
Clinical Evaluation Including Imaging
Decision Making in Lower Extremity Deformity Correction
Growth Modulation for Angular and Length Correction
Physeal Bar Excision
Acute Deformity Correction Using an Osteotomy
Gradual Deformity Correction
Hybrid Techniques for Limb Length and Deformity Correction
Motorized Intramedullary Lengthening, an Emerging Technology for Limb Length and Deformity Correction
Part II: Related Concepts and Management Options
Biomechanically Based Clinical Decision Making in Pediatric Foot and Ankle Surgery
Pediatric Joint Contractures
Physical Therapy During Limb Lengthening and Deformity Correction: Principles and Techniques
Amputation and Prosthetic Management: Amputation as a Reconstructive Option
Working in Resource-Challenged Environments
Part III: Underlying Conditions
Metabolic Disorders
Osteogenesis Imperfecta
Lower Limb Deformity in Neuromuscular Disorders: Pathophysiology, Assessment, Goals and Principles of Management
Arthrogryposis
Limb Lengthening and Deformity Correction in Patients with Skeletal Dysplasias
Lower Extremity Benign Bone Lesions and Related Conditions
Management of Juxtaphyseal Malignant Bone Tumors Around the Knee Joint: New Concepts in Limb-Sparing Surgery
Part IV: Congenital and Developmental Disorders
Congenital Femoral Deficiency Reconstruction and Lengthening Surgery
Fibular Hemimelia: Principles and Techniques of Management
Tibial Hemimelia
Congenital Pseudarthrosis of the Tibia
Controversies in Blount?s Disease
Part V: Sequelae and Complications
Methods to Enhance Bone Formation in Distraction Osteogenesis
Residual Deformities of the Hip
Post-traumatic Lower Limb Deformities in Children
Postinfectious Deformities of the Lower Limb
Iatrogenic Deformities. - ArticleOgihara T, Miyal K, Nishi K, Ishibashi K, Kumahara Y.J Clin Endocrinol Metab. 1977 Jan;44(1):91-5.An enzyme-labelled immunoassay for plasma cortisol was developed. For this alkaline-phosphatase was conjugated through 21-hemisuccinate of cortisol using water-soluble carbodiimide. An antibody was raised in rabbits against corisol-21-hemisuccinate bovine serum albumin. Before assay 10 mul samples of plasma were mixed with glutamate buffer, pH 3.3, and boiled to denature endogenous cortisol-binding globulin and alkaline-phosphatase. Separation of "bound and free" cortisol was done by the double antibody method. A linear relationship was obtained between the volume of plasma and the amount of cortisol. The minimal detectable level of plasma cortisol was 1 mug/dl and the coefficients of variation were 2.7%-4.4% (within assays) and 4.7%-6.0% (between assays). Cortisol values determined by the present method correlated well with those determined by radioimmunoassay. The present method of enzyme-labelled immunoassay is suitable for routine clinical analysis of plasma cortisol.