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  • Book
    [edited by] Peter O. Newton, Michael F. O'Brien, Harry L. Shufflebarger, Randal R. Betz, Robert A. Dickson, Jürgen Harms.
    Contents:
    History of the treatment of scoliosis
    Basic principles of scoliosis treatment
    Pathogenesis of idiopathic scoliosis
    Epidemiology of idiopathic scoliosis
    Clinical and radiographic evaluation of the scoliotic patient
    Importance of the sagittal plane: spinopelvic considerations
    Case for bracing
    Case against bracing
    Classification of adolescent idiopathic scoliosis for surgical intervention
    Biomechanics and reduction of scoliosis
    Anesthesia for scoliosis surgery
    Selective versus nonselective surgery for adolescent idiopathic scoliosis
    Selection of fusion levels
    Posterior correction techniques in late-onset scoliosis
    Use of traction in treating large scoliotic curves in idiopathic scoliosis
    Treatment of rigid adolescent idiopathic scoliosis: releases, osteotomies, and apical verterbral column resection
    Surgical treatment of the right thoracic curve pattern
    Diagnosis, treatment, and outcomes of treatment of the double thoracic curve pattern in adolescent idiopathic scoliosis
    Surgical treatment of lumbar and thoracolumbar curve patterns (Lenke Type 5): anterior versus posterior approach
    Surgical treatment of double and triple curves (Lenke Types 3,4, and 6)
    Outcomes of treatment of adolescent idiopathic scoliosis
    Complications in surgery for spinal deformity
    Spinopelvic fixation in idiopathic scoliosis
    Untreated late-onset idiopathic scoliosis and revision surgery in adults
    Osteobiological agents for spinal fusion
    Electrophysiological monitoring
    Correction without fusion
    Impact of genetics research on adolescent idiopathic scoliosis.
  • Article
    Jackson CG, Stinson JM.
    South Med J. 1979 Sep;72(9):1222-3.
    The rare occurrence of alpha 1-antitrypsin deficiency in black Americans has led to the suggestion that routine screening of such subjects in needless. Our report of a black teen-ager with deficiency of this protease inhibitor in association with status asthmaticus suggests that at least relatively young black patients with otherwise unexplained chronic pulmonary disease should be tested for this defect.
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