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  • Book
    Bernard Puech, Jean-Jacques De Laey, Graham E. Holder, editors.
    Contents:
    Introduction to Molecular Genetics and Genetic Testing for Retinal Dystrophies
    Electrophysiological Testing
    Dark Adaptation
    Fluorescein Angiography
    Indocyanine Green Angiography
    Fundus Autofluorescence Imaging in Retinal Dystrophies
    Spectral-Domain Optical Coherence Tomography in Hereditary Retinal Dystrophies
    Inherited Stationary Disorders of the Retina
    Retinitis Pigmentosa and Allied Disorders
    Leber Congenital Amaurosis and Early-Onset Retinal Dystrophy
    Retinitis Punctata Albescens
    Usher Syndromes
    Cone and Cone-Rod Dystrophies
    Enhanced S-Cone Syndrome
    Chorioretinopathies: Choroideraemia and Gyrate Atrophy
    Late-Onset Retinal Dystrophy (LORD)
    Stargardt Disease
    The Bestrophinopathies
    Retinal Dystrophies Associated with the PRPH2 Gene
    Alström Syndrome
    Bardet-Biedl Syndrome
    Cohen Syndrome
    Juvenile Neuronal Ceroid Lipofuscinoses (JNCL)
    Adult Refsum Disease (ARD)
    Abetalipoproteinemia
    LCHAD Deficiency
    Jalili Syndrome
    Spinocerebellar Ataxia 7
    Dominant Cystoid Macular Dystrophy
    Autosomal Dominant Stargardt-Like Macular Dystrophy (ELOVL4)
    Spastic Paraplegia and Retinal Degeneration: Kjellin Syndrome
    Autosomal Dominant Drusen
    Cuticular Drusen
    Extensive Macular Atrophy with Pseudodrusen-Like Appearance
    Congenital Hypotrichosis with Juvenile Macular Dystrophy
    Mitochondrial Retinopathies
    Sorsby Fundus Dystrophy
    Bietti Crystalline Corneoretinal Dystrophy
    Cystinosis
    Primary Oxalosis
    Alport Syndrome
    X-Linked Retinoschisis
    Paramacular Choriocapillaris Atrophy
    Exudative Vitreoretinopathy
    Stickler Syndrome
    Wagner Syndrome
    Incontinentia Pigmenti Type II (IP2)
    Ganglion Cell Diseases
    Pseudoxanthoma Elasticum
    Aicardi Syndrome
    Microcephaly and Chorioretinopathy With or Without Mental Retardation and Lymphedema
    Alagille Syndrome
    Future Therapies for Retinitis Pigmentosa.
    Digital Access Springer 2014
  • Article
    Carruba MO, Nisticò G, Gargiulo G.
    Ann N Y Acad Sci. 1978 Jun 12;305:242-58.
    Some behavioral effects of CNS-acting drugs have been studied in two animal species after functional impairment of central serotonergic activity. In rabbits, pretreatment with p-CPA or with 5,6-DHT counteracted the hyperthermia induced by d-amphetamine or apomorphine; the same pretreatments were ineffective in modifying pyrogen-induced fever. These data indicate a modulatory role of the 5-HT system in the hyperthermic response to dopaminergic agonists, such as d-amphetamine and apomorphine, and rule out 5-HT involvement in the hyperpyrexia induced by pyrogen. A previous intraventricular injection of 5,6-DHT significantly potentiated the increase in body temperature induced by 5-HTP in combination with a MAO inhibitor. Pretreatment with p-CPA, instead, strongly inhibited the hyperthermic response to 5-HTP. In unanesthetized fowl, at the time of the most dramatic degenerative signs of indoleaminergic neurons induced by 5,6-DHT, as documented by histofluorescence pictures, intraventricular infusion of 5-HT induced more intense behavioral, electrocortical, and body temperature modifications than in control animals. These results suggest the existence of receptor supersensitivity after 5-HT nerve ending degeneration but not after brain 5-HT depletion. The hyperthermic effect of a presynaptically acting drug, fenfluramine, was counteracted in rabbits and in fowl pretreated with either p-CPA or 5,6-DHT. Since identical behavioral, electrocortical, and body temperature effects have been observed after 5-HT or 5,6-DHT infusion into the third ventricle of fowl, it may be concluded that 5,6-DHT behaves also as a central 5-HT receptor agonist.
    Digital Access Access Options