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  • Book
    Klaus Dieter Maria Resch.
    Summary: This book adds in chapter 1 and 2 the MIN-key techniques Laser and sealing, completing the 3 MIN-key techniques of the first volume. In chapter 3 the evolution of anatomy to a key-concept of MIN is described, presenting theory and reality of anatomical perspectives that can be used by the MIN-surgeon directly in the OR. Anatomy must be elaborated according to Gestalt-Theory to become a Key of MIN. Still anatomy is the "House of Medicine", giving a mental place to all knowledge, theories and biological functions. The "surgical simulation concept" of chapter 4 as trainings environment follows, also as a key-concept of MIN. In this chapter we draw the line from Gestalt-Anatomy to a Surgical Simulation Application in Pathological Anatomy exemplified in aneurysm cases. Chapter 5 presents the best preservation technique of anatomical perishable matter, forming durable, dry and odorless specimen of unknown precision and beauty. The scientific value of this unique technique and the benefit for MIN are shown by many examples.With the Plastination gallery of chapter 6 the best head-plastinates and sheet-plastinates of head and brain complete the volume. Many of the specimen have been shown around the globe in the famous exhibitions "body worlds" (Prof. Dr. G. v. Hagens/Inventor of Plastination).

    Contents:
    Laser
    Sealing/Tachosil
    Evolution of Anatomy to a Key of MIN
    Laboratory: Surgical Simulation and Training for MIN
    The Role of Plastination for Research, Planning Strategies, Surgical Simulation and Training for MIN
    Plastination Gallery.
    Digital Access Springer 2022
  • Article
    Mashimo K.
    Scand J Infect Dis Suppl. 1978(14):172-6.
    In experimental cholecystitis in dogs, the eradication of E. coli with ampicillin, chloramphenical and kanamycin, all of which showed nearly the same MICs for this bacterial species, varied according to the differences in the penetration of the antibiotics through the liver into the bile. Antibiotics such as cephalothin and rifampicin, which are partially metabolized in the liver to inactive forms, showed higher biliary levels in CCl4-damaged animals than in the normal ones. The results were considered to be due to a reduction in the inactivating effect of esterases in the liver cells. These esterases were mainly found in the cytosol fraction and their intracellular distribution differed from that of esterases for aspirin and/or phenyl acetate.
    Digital Access Access Options