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  • Book
    Manas Das, Lee A. Baugh.
    Summary: "Thieme Test Prep for the USMLE: Medical Neuroscience by Manas Das and Lee Baugh fills a void in available board prep materials with its focus on neuroscience. Readers will learn to recall, analyze, integrate, and apply biochemical and molecular biological knowledge to solve clinical problems. Key Highlights ʺ Approximately 520 USMLE-style multiple choice questions on neuroscience, classified as easy, moderate, and difficult, with detailed explanations ʺ Questions cover clinical neurology as well as basics of neuroscience such as development, structure, and function ʺ Chapters are organized based on neuroanatomical structure and systems, from the spinal cord to the automatic nervous system ʺ Neuroimaging section and final exam chapter are invaluable tools for students to utilize before the boards ʺ Questions begin with a clinical vignette, and approximately 35% are image-based, mirroring the USMLEʼ format This essential resource will help you assess your knowledge and fully prepare for the USMLE Step 1 or COMLEX Level 1 exam"--Provided by publisher.
    Digital Access Thieme MedOne Education 2019
  • Book
    edited by A. Phillips Griffiths.
    Print 1983
  • Article
    Cunningham CM, Watson DW.
    Infect Immun. 1978 Feb;19(2):470-6.
    The effect of purified streptococcal pyrogenic exotoxins (SPE) on the antibody response to sheep erythrocytes was studied in cultures of mouse spleen cells. Purified SPE types A, B, and C shared the ability to suppress the day 4 direct plaque-forming cell response when added to cultures. SPE A and C were most suppressive at concentrations of 0.1 to 1 ng per culture, while SPE B was active at 1 microgram per culture. Pretreatment of mice with SPE A, 3 h before removal of their spleens for culture, also produced suppression. Cell populations were separated from spleens of normal and toxin-treated mice and recombined in culture to test the cellular site of action of SPE immunosuppression. When nonadherent cells (lymphocytes) and adherent cells (macrophages) from control and SPE-treated mice were separated and recombined, the plaque-forming cell response depended on the source of lymphocytes. Macrophages from toxin-treated mice functioned normally in the presence of control lymphocytes. In a further experiment, toxin pretreatment failed to suppress the plaque-forming cell response of spleen cells that were T-cell depleted and reconstituted with control thymocytes. When the T lymphocytes were removed from toxin-treated spleen cell suspensions, the remaining cells were able to respond normally to antigen if normal helper T cells were provided. The results suggest that the suppressive activity of SPE on antibody production is mediated by altered activity of T lymphocytes.
    Digital Access Access Options