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  • Book
    editors, Mitchel P. Goldman, Robert A. Weiss ; contributors, Jean-Jé́rôme Guex, Hugo Partsch, Michel R. Perrin, Albert-Adrien Ramelet, Stefano Ricci, Oscar Maleti, Marzia Lugli.
    Contents:
    1. Anatomy
    2. Adverse Sequelae and Complications of Venous Hypertension
    3. Pathophysiology of Varicose Veins
    4. Pathophysiology of Telangiectasias
    5. Noninvasive Examination of the Patient Before Sclerotherapy
    6. Use of Compression Therapy
    7. Mechanism of Action of Sclerotherapy
    8. Complications and Adverse Sequelae of Sclerotherapy
    10. Role of Surgery in the Treatment of Varicose Veins
    11. Intravascular Approaches to the Treatment of Varicose Veins : Radiofrequency, Lasers and More
    12. Clinical Methods for Sclerotherapy of Telangiectasias
    13. Treatment of Leg Telangiectasias with Laser and High-Intensity Pulsed Light
    14. Venoactive Drugs.
    Digital Access ClinicalKey 2017
  • Article
    Burgess GW, Spradbrow PB.
    Aust Vet J. 1977 Aug;53(8):363-8.
    A study of the pathogenesis of bovine ephemeral fever confirmed that the major clinical signs were fever lasting no more than 2 days, with increased respiratory rate, dyspnoea and some degree of lameness. Haematological observations revealed a neutrophilia with a left shift and a lymphopaenia at the time of peak clinical reaction. The net result was a slight leucopaenia on the day after this reaction. The most prominent pathological changes involved the lungs and synovial joints. Pulmonary emphysema and alveolar collapse with bronchiolitis, degenerative changes in synovial membranes and increased synovial fluid were observed. Specific fluorescence indicating the presence of BEF viral antigen could be detected at the time of peak clinical response in individual cells in the lungs, spleen and lymph nodes as well as neutrophils. Before and after the peak fever some fluorescence was seen in cells which appeared to be reticular cells in the lymph nodes. Viral isolation in mice could be made from blood, lungs, spleen and lymph nodes over a period of no more than 3 days. It is postulated that viral growth takes place mainly in the reticuloendothelial cells in the lungs, spleen and lymph nodes and not in vascular endothelium or lymphoid cells.
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