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  • Book
    Mitsuo Ochi, Konsei Shino, Kazunori Yasuda, Masahiro Kurosaka, editors.
    Contents:
    Part I. Anatomy and Histology of the ACL
    Chapter 1. Functional Anatomy of the ACL Fibers on the Femoral Attachment
    Chapter 2. The Anatomical Features of ACL Insertion Sites and Their Implications for Multi-Bundle Reconstruction
    Chapter 3. Discrepancy Between Macroscopic and Histological Observations
    Chapter 4. Tibial Insertion of the ACL: 3D-CT Images, Macroscopic, and Microscopic Findings
    Chapter 5. Mechanoreceptors in the ACL
    Part II. Biomechanics of the ACL
    Chapter 6. Mechanical Properties and Biomechanical Function of the ACL
    Chapter 7. Biomechanics of the Knee with Isolated One Bundle Tear of the Anterior Cruciate Ligament
    Chapter 8. Function and Biomechanics of ACL Remnant
    Chapter 9. Biomechanics of Single- and Double-Bundle ACL Reconstruction
    Chapter 10. ACL Injury Mechanisms
    Part III. Diagnostics of ACL Injury
    Chapter 11. Physical Examinations and Device Measurements for ACL Deficiency
    Chapter 12. Diagnostics of ACL Injury Using Magnetic Resonance Imaging (MRI)
    Chapter 13. Diagnosis of Injured ACL Using Three-Dimensional Computed Tomography: Usefulness for Preoperative Decision Making
    Part IV. Basic Knowledge of ACL Reconstruction
    Chapter 14. Graft Selection
    Chapter 15. Portal Placement
    Chapter 16. Femoral Bone Tunnel Placement
    Chapter 17. Tibial Bone Tunnel Placement in Double-Bundle Anterior Cruciate Ligament Reconstruction Using Hamstring Tendons
    Chapter 18. Tensioning and Fixation of the Graft
    Chapter 19. Tendon Regeneration after Harvest for ACL Reconstruction
    Chapter 20. Second-Look Arthroscopic Evaluation after ACL Reconstruction
    Chapter 21. Bone Tunnel Changes After ACL Reconstruction
    Chapter 22. Graft I impingement
    Chapter 23. Fixation Procedure
    Part V Multiple bundle ACL Reconstruction
    Chapter 24. Single- vs. Double-Bundle ACL Reconstruction
    Chapter 25. Anatomic Double-Bundle Reconstruction Procedure
    Chapter 26. Triple-Bundle ACL Reconstruction with the Semitendinosus Tendon Graft
    Part VI. ACL Augmentation
    Chapter 27. History and Advantages of ACL Augmentation
    Chapter 28. Surgical Technique of ACL Augmentation
    Part VII. ACL Reconstruction Using Bone-Patella Tendon-Bone
    Chapter 29. An Overview
    Chapter 30. Anatomical Rectangular Tunnel ACL Reconstruction with a Bone-Patellar Tendon-Bone Graft
    Chapter 31. Rectangular vs. Round Tunnel
    Part VIII. Computer-Assisted Navigation in ACL Reconstruction
    Chapter 32. Intraoperative Biomechanical Evaluation Using a Navigation System
    Chapter 33. Application of Computer-Assisted Navigation
    Part IV. ACL Injury in Patients with Open Physes
    Chapter 34. ACL Reconstruction with Open Physes
    Chapter 35. Avulsion Fracture of the ACL
    Part V. Revision ACL Reconstruction
    Chapter 36. Double-Bundle Technique
    Chapter 37. Bone-Patellar Tendon-Bone Graft via Round Tunnel
    Chapter 38. Anatomical Revision ACL Reconstruction with Rectangular Tunnel Technique
    Chapter 39. One- vs. Two-Stage Revision Anterior Cruciate Ligament Reconstruction
    Part VI. Complications of ACL Reconstruction
    Chapter 40. Complications of ACL Reconstruction
    Part VII. Future of ACL Reconstruction
    Chapter 41. Future Challenges of Anterior Cruciate Ligament Reconstruction: Biological Modulation Using a Growth Factor Application for Enhancement of Graft Healing
    Chapter 42. Strategies to Enhance Biological Tendon-Bobe Healing in Anterior Cruciate Ligament Reconstruction
    Chapter 43. Tissue engineering approach for ACL healing.
    Digital Access Springer 2016
  • Article
    Vicuna R, Ikeda JE, Hurwitz J.
    J Biol Chem. 1977 Apr 25;252(8):2534-44.
    In the presence of RNA polymerase, RNase H, discriminatory factors alpha and beta, Escherichia coli binding protein, DNA elongation factor I, DNA elongation factor II preparation, DNA polymerase III, and ATP, UTP, GTP, CTP, dATP, dTTP, dGTP, and dCTP, fd viral DNA can be quantitatively converted to RFII containing a unique gap in the linear minus strand. This gap, mapped with the aid of restriction endonucleases HinII and HpaII, is located within Fragment Hpa-H of the fd genome. The discrimination reaction has been resolved into two steps: Step A, fd viral DNA, E. coli binding protein, and discriminatory factors alpha and beta form a protein DNA complex; Step B, the complex isolated by agarose gel filtration selectively forms fd RFII when supplemented with RNase H, RNA polymerase, and the DNA elongation proteins. The omission of any of the proteins described above during the first reaction resulted in either no discrimination or a decrease in discrimination when the missing protein was added during the second step. Results are presented which indicate that E. coli binding protein, discriminatory factors alpha and beta, and RNase H must be present during the time RNA synthesis occurs in order to selectively form RFII from fd DNA and not phiX RFII. The amount of fd and phiX174 RNA-DNA hybrid formed in vitro is directly related to the DNA synthesis observed. Thus, under discriminatory conditions, only fd viral DNA leads to fd RNA-DNA complexes and no phiX RNA-DNA hybrid is formed. Under nondiscriminatory conditions, both DNAs yield RNA-DNA hybrids and DNA synthesis. In the absence of discriminatory factor alpha, no RNA-DNA hybrid is formed with either DNA, and in turn, no DNA synthesis is detected with either DNA template.
    Digital Access Access Options