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  • Book
    Pasquale Perrone Filardi, editor.
    Summary: This book summarizes how the renin angiotensin system is implicated in the progression of atherosclerotic disease as well as of left ventricular dysfunction and reviews the action of angiotensin converting enzyme inhibitors and AT1 receptor antagonists on reducing morbidity and mortality in patients with left ventricular dysfunction or in those at high cardiovascular risk with preserved ventricular function. ACEi and ARBS in Hypertension and Heart Failure is a highly practical reference reviewing the evidence and providing a rationale for the appropriate use of RAS antagonists in cardiovascular diseases. It is written in an easy-to-follow format and with many illustrations to aid clarity and the assimilation of information. Each chapter is written by established authorities in their fields who are also experienced in explaining often complex concepts. The result is a unique book which is not only comprehensive but also clear and useful for the busy medical practitioner.

    Contents:
    Pathogenetic mechanisms of RAS involvement in cardiovascular diseases
    Antiotensin converting enzyme inhibitors and at1 antagonists for treatment of hypertension.- Renin angiotensin system antagonism in heart failure
    Impact of co-morbidities on RAS inhibitors choice in hypertensive and heart failure patients
    Target organ damage and RAS blockade.
    Digital Access Springer 2015
  • Article
    Uhr JW, Vitetta ES.
    Natl Cancer Inst Monogr. 1978 May(48):295-300.
    Murine lymphocytes were enzymatically radioiodinated and the class of immunoglobulin (isotype) on the cell surface was studied as a function of differentiation and ontogeny. The results indicate that IgM is the first isotype to appear and that IgD is subsequently acquired. A proportion of IgM-bearing cells also bear IgD. The acquisition of IgD does not appear to be under the influence of the thymus or of exposure to antigen. The observations suggest a sequence of differentiation steps in which B-cells first express IgM then acquire IgD (to become "double bearers"), and eventually lose IgM. The relationship of the IgD-bearing cells to the IgG-bearing memory cells has not yet been established. The implications of these findings with regard to function and genetic organization are discussed.
    Digital Access Access Options