BookLee Yee-Ki, Siu Chung-Wah.
Contents:
Calcium Handling in hiPSC-Derived Cardiomyocytes
Myocardial Infarction and Heart Failure
Embryonic Stem Cells/Pluripotent Stem Cells
Human Induced Pluripotent Stem Cell (hiPSC)-Derived Cardiomyocytes as a Source for Cardiac Regeneration
Spontaneous Cardiac Differentiation
Mesoderm Enrichment by Wnt-Signaling Growth Factors
Endoderm-Like Cell (END)-2 Co-culture Method
Calcium Homeostasis in Cardiomyocytes
Sarcoplasmic Reticulum (SR) Governs Maturity of Cardiomyocytes
SR Junctional Proteins Play a Role in Calcium Flux Between Cytosol and SR
Spatial and Temporal Ca2+ Wavefront Dictated by T-Tubule Structure
IP3-Mediated Calcium Release Contributes to Whole-Cell Calcium Transients
Calcium Handling Properties of hES-Derived Cardiomyocytes
Calcium Handling Properties of hiPSC-Derived Cardiomyocytes
iPSC-Derived Cardiomyocyte as a Potential Platform for Disease Modeling of the Impaired Calcium Handling-Related Syndrome
Disease Modeling of Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT)
Mitochondrial Dysfunction-Related Cardiomyopathy-Impaired SR Ca2+ Reuptake
Measurement of Cytosolic [Ca2+] by Fluorescence Confocal Microscopy
Materials
Calibration of Fluo-3
Isolation of hiPSC- and hESC-Derived Cardiomyocytes
Recording of Cytosolic [Ca2+]
RT-qPCR of Calcium Handling Proteins.