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  • Book
    William L. Weston, Alfred T. Lane, Joseph G. Morelli.
    Contents:
    1. Structure and function of the skin
    2. Evaluation of children with skin disease
    3. Acne
    4. Dermatitis
    5. Bacterial infections (Pyodermas) and spirochetal infections of the skin
    6. Fungal and yeast infections of the skin
    7. Infestations
    8. Viral infections
    9. Papulosquamous disorders
    10. Sun sensitivity
    11. Bullous diseases and mucocutaneous syndromes
    12. Skin cysts and nodules
    13. Vascular lesions
    14. Vascular reactions: urticaria, erythemas, and purpuras
    15. Hair disorders
    16. Nail disorders
    17. Disorders of pigmentation: the white lesions and the brown lesions
    18. Immobile and hypermobile skin
    19. Genodermatoses
    20. Drug eruptions
    21. Skin diseases in newborns
    22. Dermatopharmacology and topical formulary.
    Digital Access ScienceDirect 2007
    Print Access Request
    Location
    Version
    Call Number
    Items
    Books: General Collection (Downstairs)
    2007
    RJ511 .W46 2007
    1
  • Article
    Tanaka N, Tanaka H, Toyama Y, Kashima T, Niimura T.
    Am Heart J. 1975 Sep;90(3):369-75.
    Three patients with aortitis syndrome ehibited paroxysmal hypertension which seemed to result from baroreceptor dysfunction. All of the patients had signs of active inflammation of aortitis syndrome and stenotic carotid and subclavian arteries. During the attacks, the blood pressure rose to at least 230 mm. Hg systolic and the heart rate exceeded 100. However, with prolonged administration of steroid hormones, the attacks ceased. In two patients with dilated thoracic aortas and aortic regurgitation, the attacks of paroxysmal hypertension occurred without apparent precipitating factors and were followed by anginal pain with marked ST depression. The sympathicotonic state resulting from the disturbance of the baroreceptors was considered to be responsible for the attacks. In another patient, the attacks occurred in the course of treatment with a steroid hormone and were provoked only by voluntary micturition. This post-micturition hypertension was presumed to be an expression of abnormal overshooting following a fall in blood pressure after voiding.
    Digital Access Access Options